Journal of Parkinson's Disease

Journal of Parkinson’s Disease

ISSN
1877-7171
Volume
1; 4 issues
Status
Last issue (1:4) published on 29 December 2011
Next issue
2:1 scheduled for March 2012
Website
www.journalofparkinsonsdisease.com
Subject
Neurosciences
Institutional subscription for 2012 €555 / US$776 Incl. postage and handling
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The Journal of Parkinson’s Disease is dedicated to providing an open forum for original research in basic science, translational research and clinical medicine that will expedite our fundamental understanding and improve treatment of Parkinson’s disease. The journal is international and multidisciplinary and aims to promote progress in the epidemiology, etiology, genetics, molecular correlates, pathogenesis, pharmacology, psychology, diagnosis and treatment of Parkinson’s disease. It will publish research reports, reviews, short communications, and letters-to-the-editor and offers very rapid publication and an affordable open access option.


Click here to see: Editor-in-Chief Patrik Brundin introduces the Journal of Parkinson’s Disease.


 


 


 


 


 


 

Editors-in-Chief

Patrik Brundin, MD, PhD
Van Andel Research Institute
Grand Rapids, MI
USA
Wallenberg Neuroscience Center
Lund University
Lund
Sweden
Email: patrik.brundin@iospress.com

J. William Langston, MD
The Parkinson's Institute and Clinical Center
Sunnyvale, CA
USA
Email: bill.langston@iospress.com

Associate Editor for Reviews

M. Angela Cenci
Sweden

Associate Editors

Bastiaan Bloem
The Netherlands

Mark Cookson
USA

Dennis Dickson
USA

Robert Edwards
USA

Howard Federoff
USA

Thomas Gasser
Germany

Glenda Halliday
Australia

Jan Petter Larsen
Norway

Andres Lozano
Canada

Tamas Revesz
United Kingdom

Nick Wood
United Kingdom

Editorial Board

Dag Aarsland, Norway
Patrick Aebischer, Switzerland
Yves Agid, France
Ernest Arenas, Sweden
Alberto Ascherio, USA
Roger Barker, United Kingdom
Alim Benabid, France
Hagai Bergman, Israel
Anders Bjorklund, Sweden
Vincenzo Bonifati, The Netherlands
Heiko Braak, Germany
Alexis Brice, France
David J. Brooks, United Kingdom
Robert Burke, USA
Paolo Calabresi, Italy
Piu Chan, China
K. Ray Chaudhuri, United Kingdom
Marie Francoise Chesselet, USA
Cynthia Comella, USA
Ted M. Dawson, USA
Omar M. El-Agnaf, United Arab Emirates
Stanley Fahn, USA
Matt Farrer, Canada
Joaquim Ferreira, Portugal
Nir Giladi, Israel
Ann M. Graybiel, USA
J. Timothy Greenamyre, USA
James G. Greene, USA
John Hardy, United Kingdom
Nobutaka Hattori, Japan
Etienne C. Hirsch, France
Joseph Jankovic, USA
Christine Klein, Germany
Jeffrey H. Kordower, USA
Seung-Jae Lee, Korea
Virginia Lee, USA
Laura Marsh, USA
Eldad Melamed, Israel
Alice Nieuwboer, Belgium
Robert Nussbaum, USA
Ronald F. Pfeiffer, USA
Pierre Pollak, Switzerland
Serge Przedborski, USA
Niall Quinn, United Kingdom
Heinz Reichmann, Germany
Peter Riederer, Germany
Trevor Robbins, United Kingdom
Anthony Schapira, United Kingdom
Todd Sherer, USA
Ira Shoulson, USA
Dennis Steindler, USA
Gerald Stern, United Kingdom
Fabrizio Stocchi, Italy
Jun Takahashi, Japan
Eng King Tan, Singapore
Caroline M. Tanner, USA
Eduardo Tolosa, Spain
John Q. Trojanowski, USA

See instructions to authors.

Embase

Scientists Report First Step in Strategy for Cell Replacement Therapy in Parkinson’s Disease

25 Jan 2012 - Induced pluripotent stem cells (iPSC) are a promising avenue for cell replacement therapy in neurologic diseases. For example, mouse and human iPSCs have been used to generate dopaminergic (DA) neurons that improve symptoms in rat Parkinson’s disease models. Reporting in the current issue of the Journal of Parkinson’s Disease, a group of scientists from Japan evaluated the growth, differentiat...

New Research Reveals How α-Synuclein Interacts with Cell Membranes in Parkinson’s Disease

19 Jan 2012 - The accumulation of α-synuclein, a small, negatively charged protein, in neural cells, is one of the hallmarks of Parkinson’s disease. It has been suggested that oligomeric α-synuclein causes membranes to become permeable, or to form channels on the outer cell membrane. Now, a group of scientists from Sweden has found a way to reliably replicate α-synuclein aggregation on cell membranes to ...

New Study Supports View that Lewy Bodies Are Not the Primary Cause of Cell Death in Parkinson’s Disease

10 Jan 2012 - The pathology of Parkinson’s disease is characterized by a loss of dopamine-producing neurons in the pars compacta of the substantia nigra (SN), an area of the brain associated with motor control, along with the development of α-synuclein (αS) protein in the form of Lewy bodies (LB) in the neurons that survive. The spread of LB pathology is thought to progress along with the clinical course o...

Genetic Factors Can Predict the Progression of Parkinson’s Disease

16 Dec 2011 - Parkinson’s disease is marked by the abnormal accumulation of α-synuclein and the early loss of dopamine neurons in the substantia nigra region of the brain. A polymorphism in the promotor of α-synuclein gene known as NACP-Rep1 has been implicated as a risk factor for the disease. Now, researchers have found that different variants of NACP-Rep1 and its interaction with the microtubule-associa...

New Study First to Link Mitochondrial Dysfunction and alpha-Synuclein Multiplication in Human Fibroblasts

07 Oct 2011 - A new study in the Journal of Parkinson’s Disease shows for the first time the effects of α-Synuclein (α-syn) gene multiplication on mitochondrial function and susceptibility to oxidative stress in human tissue. Mitochondrial dysfunction has been frequently implicated in the neurodegenerative process that underlies Parkinson’s disease, but the basis for this has not been fully understood. ...

Ability to Ride a Bike Can Aid Differential Diagnosis of Parkinson’s Disease in Any Setting

07 Oct 2011 - In a new study published today in the Journal of Parkinson’s Disease, Japanese researchers report that the ability to ride a bike can differentiate between atypical parkinsonism and Parkinson’s disease, regardless of the environment or situations for bicycling. ...